BRE over-expression promotes growth of hepatocellular carcinoma
- 24 December 2009
- journal article
- research article
- Published by Elsevier in Biochemical and Biophysical Research Communications
- Vol. 391 (3) , 1522-1525
Abstract
No abstract availableKeywords
Funding Information
- Hong Kong Research Grants Council Direct Allocation to the Chinese University of Hong Kong, and by a Competitive Earmarked Research (2041332)
- Research Grant Committee (CUHK463708)
This publication has 19 references indexed in Scilit:
- RAP80 Targets BRCA1 to Specific Ubiquitin Structures at DNA Damage SitesScience, 2007
- Ubiquitin-Binding Protein RAP80 Mediates BRCA1-Dependent DNA Damage ResponseScience, 2007
- A Death Receptor-associated Anti-apoptotic Protein, BRE, Inhibits Mitochondrial Apoptotic PathwayJournal of Biological Chemistry, 2004
- Regulation of BRCC, a Holoenzyme Complex Containing BRCA1 and BRCA2, by a Signalosome-like Subunit and Its Role in DNA RepairMolecular Cell, 2003
- Expression of Human BRE in Multiple IsoformsBiochemical and Biophysical Research Communications, 2001
- Bid-deficient mice are resistant to Fas-induced hepatocellular apoptosisNature, 1999
- BRE: a modulator of TNF‐α actionThe FASEB Journal, 1998
- Two CD95 (APO‐1/Fas) signaling pathwaysThe EMBO Journal, 1998
- Evidence against a functional site for Bcl-2 downstream of caspase cascade in preventing apoptosisOncogene, 1997
- Bcl-2 Prevents CD95 (Fas/APO-1)-induced Degradation of Lamin B and Poly(ADP-ribose) Polymerase and Restores the NF-κB Signaling PathwayJournal of Biological Chemistry, 1996