The peptidyl-prolyl isomerase, Pin1, facilitates NF-κB binding in hepatocytes and protects against hepatic ischemia/reperfusion injury
Authors:
- Nozomu SakaiNSNozomu Sakai
- Callisia ClarkeCCCallisia Clarke
- Rebecca SchusterRSRebecca Schuster
- John BlanchardJBJohn Blanchard
- Michael J. EdwardsMEMichael J. Edwards
- Alex B. LentschALAlex B. Lentsch
- 1 August 2009
- journal article
- Published by Elsevier in Journal of Hepatology
- Vol. 51 (2) , 296-306
Abstract
No abstract availableKeywords
This publication has 35 references indexed in Scilit:
- Roles of nuclear factor‐κB in postischemic liverHepatology Research, 2007
- A critical role for Pin1 in allergic pulmonary eosinophilia in ratsJournal of Allergy and Clinical Immunology, 2007
- Hepatocyte NF-κB activation is hepatoprotective during ischemia-reperfusion injury and is augmented by ischemic hypothermiaAmerican Journal of Physiology-Gastrointestinal and Liver Physiology, 2007
- PIN1 expression contributes to hepatic carcinogenesisThe Journal of Pathology, 2006
- REVIEW: Pin1 in Alzheimer's diseaseJournal of Neurochemistry, 2006
- Regulation of NF-κB Signaling by Pin1-Dependent Prolyl Isomerization and Ubiquitin-Mediated Proteolysis of p65/RelAMolecular Cell, 2003
- Inflammatory mechanisms and therapeutic strategies for warm hepatic ischemia/reperfusion injuryHepatology, 2000
- Function of WW Domains as Phosphoserine- or Phosphothreonine-Binding ModulesScience, 1999
- Role of tumor necrosis factor-alpha in the pathophysiologic alterations after hepatic ischemia/reperfusion injury in the rat.Journal of Clinical Investigation, 1990
- Improved method for quantification of tissue PMN accumulation measured by myeloperoxidase activityJournal of Pharmacological Methods, 1990