The Mitochondrial ATPase Inhibitory Factor 1 Triggers a ROS-Mediated Retrograde Prosurvival and Proliferative Response
Authors:
- Laura FormentiniLaura Formentini
- María Sánchez-AragóMSMaría Sánchez-Aragó
- Laura Sánchez-CenizoLSLaura Sánchez-Cenizo
- José M. CuezvaJosé M. Cuezva
- 1 March 2012
- journal article
- Published by Elsevier in Molecular Cell
- Vol. 45 (6) , 731-742
Abstract
No abstract availableKeywords
This publication has 48 references indexed in Scilit:
- Reductive carboxylation supports growth in tumour cells with defective mitochondriaNature, 2011
- NF-κB controls energy homeostasis and metabolic adaptation by upregulating mitochondrial respirationNature Cell Biology, 2011
- Hallmarks of Cancer: The Next GenerationCell, 2011
- Regulation of cancer cell metabolismNature Reviews Cancer, 2011
- Crosstalk of reactive oxygen species and NF-κB signalingCell Research, 2010
- Mitochondrial reactive oxygen species regulate cellular signaling and dictate biological outcomesTrends in Biochemical Sciences, 2010
- Role of calcineurin, hnRNPA2 and Akt in mitochondrial respiratory stress-mediated transcription activation of nuclear gene targetsBiochimica et Biophysica Acta (BBA) - Bioenergetics, 2010
- IEX-1 targets mitochondrial F1Fo-ATPase inhibitor for degradationCell Death & Differentiation, 2008
- Dichloroacetate (DCA) as a potential metabolic-targeting therapy for cancerBritish Journal of Cancer, 2008
- Nuclear factor-κB in cancer development and progressionNature, 2006