The Adaptor Protein CARD9 Protects against Colon Cancer by Restricting Mycobiota-Mediated Expansion of Myeloid-Derived Suppressor Cells
- 1 September 2018
- Vol. 49 (3) , 504-514.e4
Abstract
No abstract availableKeywords
Funding Information
- National Natural Science Foundation of China (81572354, 81772542, 81630058, 91542107)
- National Institutes of Health (AI116722)
- Center for Inflammation and Cancer in MDACC
- Natural Science Foundation of Jiangsu Province in China (BK20161400)
- Top six talent project in Jiangsu Province (WSW-031)
This publication has 60 references indexed in Scilit:
- Card9‐dependent IL‐1β regulates IL‐22 production from group 3 innate lymphoid cells and promotes colitis‐associated cancerEuropean Journal of Immunology, 2017
- Regulatory T-cell Response to EnterotoxigenicBacteroides fragilisColonization Triggers IL17-Dependent Colon CarcinogenesisCancer Discovery, 2015
- Dysbiosis of Fungal Microbiota in the Intestinal Mucosa of Patients with Colorectal AdenomasScientific Reports, 2015
- Fungal inflammatory masses masquerading as colorectal cancer: a case reportBMC Research Notes, 2015
- C-type Lectin Receptor Dectin-3 Mediates Trehalose 6,6′-Dimycolate (TDM)-induced Mincle Expression through CARD9/Bcl10/MALT1-dependent Nuclear Factor (NF)-κB ActivationJournal of Biological Chemistry, 2014
- Card9 Mediates Intestinal Epithelial Cell Restitution, T-Helper 17 Responses, and Control of Bacterial Infection in MiceGastroenterology, 2013
- The adaptor protein CARD9 is essential for the activation of myeloid cells through ITAM-associated and Toll-like receptorsNature Immunology, 2007
- Dectin-1 is required for host defense against Pneumocystis carinii but not against Candida albicansNature Immunology, 2006
- Yeast metabolic products, yeast antigens and yeasts as possible triggers for irritable bowel syndromeEuropean Journal of Gastroenterology & Hepatology, 2005
- Severity of inflammation is a risk factor for colorectal neoplasia in ulcerative colitisGastroenterology, 2004