Oxidative stress and Nrf2 signaling in McArdle disease
Authors:
- Yu KitaokaYKYu Kitaoka
- Daniel I. OgbornDODaniel I. Ogborn
- Mats I. NilssonMNMats I. Nilsson
- Nicholas J. MocellinNMNicholas J. Mocellin
- Lauren G. MacNeilLMLauren G. MacNeil
- Mark A. TarnopolskyMTMark A. Tarnopolsky
- 1 November 2013
- journal article
- research article
- Published by Elsevier in Molecular Genetics and Metabolism
- Vol. 110 (3) , 297-302
Abstract
No abstract availableKeywords
This publication has 29 references indexed in Scilit:
- Monocarboxylate transporters and mitochondrial creatine kinase protein content in McArdle diseaseMolecular Genetics and Metabolism, 2013
- Acute exercise stress activates Nrf2/ARE signaling and promotes antioxidant mechanisms in the myocardiumFree Radical Biology and Medicine, 2012
- Six novel mutations in the myophosphorylase gene in patients with McArdle disease and a family with pseudo-dominant inheritance patternMolecular Genetics and Metabolism, 2011
- Dysfunctional Nrf2–Keap1 redox signaling in skeletal muscle of the sedentary oldFree Radical Biology and Medicine, 2010
- Nrf2:INrf2 (Keap1) signaling in oxidative stressFree Radical Biology and Medicine, 2009
- Nrf2, a multi‐organ protector?The FASEB Journal, 2005
- Nrf2–Keap1 defines a physiologically important stress response mechanismTrends in Molecular Medicine, 2004
- Breakdown of adenine nucleotide pool in fatiguing skeletal muscle in McArdle's disease: A noninvasive 31P‐MRS and EMG studyMuscle & Nerve, 2003
- Excess purine degradation in exercising muscles of patients with glycogen storage disease types V and VII.Journal of Clinical Investigation, 1985
- Hypoxanthine and McArdle disease: A clue to metabolic stress in the working forearmMuscle & Nerve, 1983