Mineralocorticoid and angiotensin II type 1 receptors in the subfornical organ mediate angiotensin II – induced hypothalamic reactive oxygen species and hypertension
- 1 August 2016
- journal article
- Published by Elsevier in Neuroscience
- Vol. 329, 112-121
Abstract
No abstract availableKeywords
This publication has 34 references indexed in Scilit:
- Knockdown of mineralocorticoid or angiotensin II type 1 receptor gene expression in the paraventricular nucleus prevents angiotensin II hypertension in ratsThe Journal of Physiology, 2014
- Angiotensin-Generated Reactive Oxygen Species in Brain and Pathogenesis of Cardiovascular DiseasesAntioxidants & Redox Signaling, 2013
- Central Mineralocorticoid Receptors and the Role of Angiotensin II and Glutamate in the Paraventricular Nucleus of Rats With Angiotensin II–Induced HypertensionHypertension, 2013
- Central neuromodulatory pathways regulating sympathetic activity in hypertensionJournal of Applied Physiology, 2012
- Angiotensin-II-induced reactive oxygen species along the SFO-PVN-RVLM pathway: implications in neurogenic hypertensionBrazilian Journal of Medical and Biological Research, 2011
- Central neuronal activation and pressor responses induced by circulating ANG II: role of the brain aldosterone-“ouabain” pathwayAmerican Journal of Physiology-Heart and Circulatory Physiology, 2010
- Angiotensinergic Regulation of Autonomic and Neuroendocrine Outputs: Critical Roles for the Subfornical Organ and Paraventricular NucleusNeuroendocrinology, 2009
- Distribution of epithelial sodium channels and mineralocorticoid receptors in cardiovascular regulatory centers in rat brainAmerican Journal of Physiology-Regulatory, Integrative and Comparative Physiology, 2005
- NADPH Oxidase–Derived Superoxide Anion Mediates Angiotensin II–Induced Pressor Effect via Activation of p38 Mitogen–Activated Protein Kinase in the Rostral Ventrolateral MedullaCirculation Research, 2005
- CHRONIC EFFECTS OF ANGIOTENSIN II and AT1 RECEPTOR ANTAGONISTS IN SUBFORNICAL ORGAN‐LESIONED RATSClinical and Experimental Pharmacology and Physiology, 2005