Impaired function of cerebral parenchymal arterioles in experimental preeclampsia
Authors:
- 1 September 2018
- journal article
- research article
- Published by Elsevier in Microvascular Research
- Vol. 119, 64-72
Abstract
No abstract availableKeywords
Funding Information
- NIH National Institute of Neurological Disorders and Stroke (R01 NS045940)
- Preeclampsia Foundation
- The Totman Medical Research Trust
- The Cardiovascular Research Institute of Vermont
This publication has 47 references indexed in Scilit:
- Relaxin causes selective outward remodeling of brain parenchymal arterioles via activation of peroxisome proliferator‐activated receptor‐γThe FASEB Journal, 2011
- Cerebral vascular adaptation to pregnancy and its role in the neurological complications of eclampsiaJournal of Applied Physiology, 2011
- Pericytes in capillaries are contractile in vivo, but arterioles mediate functional hyperemia in the mouse brainProceedings of the National Academy of Sciences, 2010
- Astrocytic endfoot Ca 2+ and BK channels determine both arteriolar dilation and constrictionProceedings of the National Academy of Sciences, 2010
- SK Ca and IK Ca Channels, Myogenic Tone, and Vasodilator Responses in Middle Cerebral Arteries and Parenchymal ArteriolesStroke, 2009
- Increased Lectin-Like Oxidized Low-Density Lipoprotein Receptor-1 Expression in the Maternal Vasculature of Women With PreeclampsiaHypertension, 2009
- Progression of Cerebral Small Vessel Disease in Relation to Risk Factors and Cognitive ConsequencesStroke, 2008
- Self-reported cognitive functioning in formerly eclamptic womenAmerican Journal of Obstetrics and Gynecology, 2007
- White Matter Lesions in an Unselected Cohort of the ElderlyStroke, 2006
- Changes in LDL size and HDL concentration in normal and preeclamptic pregnanciesAtherosclerosis, 2002