Spinacetin alleviates doxorubicin-induced cardiotoxicity by initiating protective autophagy through SIRT3/AMPK/mTOR pathways
Authors:
- 12 April 2022
- journal article
- research article
- Published by Elsevier in Phytomedicine
- Vol. 101, 154098
Abstract
No abstract availableKeywords
This publication has 34 references indexed in Scilit:
- Identification of AMPK Phosphorylation Sites Reveals a Network of Proteins Involved in Cell Invasion and Facilitates Large-Scale Substrate PredictionCell Metabolism, 2015
- SIRT3 deacetylates FOXO3 to protect mitochondria against oxidative damageFree Radical Biology and Medicine, 2013
- The NAD-dependent deacetylase SIRT2 is required for programmed necrosisNature, 2012
- Identification of the molecular basis of doxorubicin-induced cardiotoxicityNature Medicine, 2012
- Cardiovascular toxicity induced by chemotherapy, targeted agents and radiotherapy: ESMO Clinical Practice GuidelinesAnnals of Oncology, 2012
- Doxorubicin-induced cardiomyopathy: From molecular mechanisms to therapeutic strategiesJournal of Molecular and Cellular Cardiology, 2012
- The NAD+ Precursor Nicotinamide Riboside Enhances Oxidative Metabolism and Protects against High-Fat Diet-Induced ObesityCell Metabolism, 2012
- Canonical and non-canonical autophagy: variations on a common theme of self-eating?Nature Reviews Molecular Cell Biology, 2011
- The SirT3 Divining Rod Points to Oxidative StressMolecular Cell, 2011
- The sources of pesticide contamination in Queensland livestockAustralian Veterinary Journal, 1990