T-2 toxin induces cardiac fibrosis by causing metabolic disorders and up-regulating Sirt3/FoxO3α/MnSOD signaling pathway-mediated oxidative stress
- 16 March 2024
- journal article
- research article
- Published by Elsevier in Journal of Environmental Sciences
- Vol. 150, 532-544
Abstract
No abstract availableKeywords
Funding Information
- Xi’an Jiaotong University
- National Natural Science Foundation of China (81872567)
This publication has 41 references indexed in Scilit:
- Effects of histidine load on ammonia, amino acid, and adenine nucleotide concentrations in ratsAmino Acids, 2019
- Neuroprotective role of hyperforin on aluminum maltolate-induced oxidative damage and apoptosis in PC12 cells and SH-SY5Y cellsChemico-Biological Interactions, 2019
- Selenium deficiency aggravates T-2 toxin-induced injury of primary neonatal rat cardiomyocytes through ER stressChemico-Biological Interactions, 2018
- Acetylation control of cardiac fatty acid β-oxidation and energy metabolism in obesity, diabetes, and heart failureBiochimica et Biophysica Acta (BBA) - Molecular Basis of Disease, 2016
- Prevalence of Selenium, T-2 Toxin, and Deoxynivalenol in Kashin–Beck Disease Areas in Qinghai Province, Northwest ChinaBiological Trace Element Research, 2015
- Oxidative stress-mediated cytotoxicity and metabolism of T-2 toxin and deoxynivalenol in animals and humans: an updateArchives of Toxicology, 2014
- Mitochondria, Oxidants, and AgingCell, 2005
- Homocysteine and Risk of Ischemic Heart Disease and StrokeJAMA, 2002
- An assay for superoxide dismutase activity in mammalian tissue homogenatesAnalytical Biochemistry, 1989
- Elevation of thiobarbituric acid values in the rat liver intoxicated by T-2 toxinBiochemical Medicine, 1984