Lack of correlation between repair of DNA interstrand cross-links and hypersensitivity of hamster cells towards mitomycin C and cisplatin.
- 16 October 1998
- journal article
- Published by Wiley in FEBS Letters
- Vol. 437 (1) , 97-100
Abstract
No abstract availableKeywords
This publication has 14 references indexed in Scilit:
- Genetic analysis of mitomycin C-hypersensitive Chinese hamster cell mutantsMutagenesis, 1994
- Role of the human ERCC-1 gene in gene-specific repair of cisplatin-induced DNA damageNucleic Acids Research, 1994
- Genetic diversity of mitomycin C-hypersensitive Chinese hamster cell mutants: A new complementation group with chromosomal instabilitySomatic Cell and Molecular Genetics, 1993
- An intrastrand d(GpG) platinum crosslink in duplex M13 DNA is refractory to repair by human cell extracts.Proceedings of the National Academy of Sciences, 1992
- The Chinese hamster cell mutant V-H4 is homologous to Fanconi anemia (complementation group A)Cytogenetic and Genome Research, 1991
- The Chinese hamster V79 cell mutant V-H4 is phenotypically like Fanconi anemia cellsSomatic Cell and Molecular Genetics, 1990
- A simple salting out procedure for extracting DNA from human nucleated cellsNucleic Acids Research, 1988
- Processing of psoralen adducts in an active human gene: Repair and replication of DNA containing monoadducts and interstrand cross-linksCell, 1987
- Adducts of the antitumor drug cis-diamminedichloroplatinum(II) with DNA: formation, identification, and quantitationBiochemistry, 1985
- Reductive metabolism and alkylating activity of mitomycin C induced by rat liver microsomesBiochemistry, 1981