A Novel Motif in the Crohn's Disease Susceptibility Protein, NOD2, Allows TRAF4 to Down-regulate Innate Immune Responses

This publication has 41 references indexed in Scilit:

• Inhibition of RIP2's tyrosine kinase activity limits NOD2-driven cytokine responses
  Genes & Development, 2010
• ATG16L1 and NOD2 Interact in an Autophagy-Dependent Antibacterial Pathway Implicated in Crohn's Disease Pathogenesis
  Gastroenterology, 2010
• MDP-NOD2 stimulation induces HNP-1 secretion, which contributes to NOD2 antibacterial function
  Inflammatory Bowel Diseases, 2010
• Nod2 is required for the regulation of commensal microbiota in the intestine
  Proceedings of the National Academy of Sciences, 2009
• ITCH K63-Ubiquitinates the NOD2 Binding Protein, RIP2, to Influence Inflammatory Signaling Pathways
  Current Biology, 2009
• MEKK4 Sequesters RIP2 to Dictate NOD2 Signal Specificity
  Current Biology, 2008
• The Ubiquitin-Editing Enzyme A20 Restricts Nucleotide-Binding Oligomerization Domain Containing 2-Triggered Signals
  Immunity, 2008
• IκB Kinase β Phosphorylates the K63 Deubiquitinase A20 To Cause Feedback Inhibition of the NF-κB Pathway
  Molecular and Cellular Biology, 2007
• Molecular mechanisms involved in the regulation of cytokine production by muramyl dipeptide
  Biochemical Journal, 2007
• The Crohn's Disease Protein, NOD2, Requires RIP2 in Order to Induce Ubiquitinylation of a Novel Site on NEMO
  Current Biology, 2004