Sudden infant death syndrome–associated mutations in the sodium channel beta subunits

This publication has 35 references indexed in Scilit:

• Loss-of-function mutation of the SCN3B-encoded sodium channel 3 subunit associated with a case of idiopathic ventricular fibrillation
  Cardiovascular Research, 2009
• α1-Syntrophin Mutations Identified in Sudden Infant Death Syndrome Cause an Increase in Late Cardiac Sodium Current
  Circulation: Arrhythmia and Electrophysiology, 2009
• A Mutation in the β3 Subunit of the Cardiac Sodium Channel Associated With Brugada ECG Phenotype
  Circulation: Cardiovascular Genetics, 2009
• Mutations in Sodium Channel β1- and β2-Subunits Associated With Atrial Fibrillation
  Circulation: Arrhythmia and Electrophysiology, 2009
• Syntrophin mutation associated with long QT syndrome through activation of the nNOS–SCN5A macromolecular complex
  Proceedings of the National Academy of Sciences, 2008
• Molecular and Functional Characterization of Novel Glycerol-3-Phosphate Dehydrogenase 1–Like Gene ( GPD1-L ) Mutations in Sudden Infant Death Syndrome
  Circulation, 2007
• Mutation in Glycerol-3-Phosphate Dehydrogenase 1–Like Gene ( GPD1-L ) Decreases Cardiac Na ⁺ Current and Causes Inherited Arrhythmias
  Circulation, 2007
• SCN4B -Encoded Sodium Channel β4 Subunit in Congenital Long-QT Syndrome
  Circulation, 2007
• A mechanism for sudden infant death syndrome (SIDS): Stress-induced leak via ryanodine receptors
  Heart Rhythm, 2007
• Novel mechanism for sudden infant death syndrome: Persistent late sodium current secondary to mutations in caveolin-3
  Heart Rhythm, 2007