Paradoxical E-cadherin increase in 5FU-resistant colon cancer is unaffected during mesenchymal–epithelial reversion induced by γ-secretase inhibition

This publication has 48 references indexed in Scilit:

• Metastatic Progression of Prostate Cancer and E-Cadherin
  The American Journal of Pathology, 2011
• Notch signaling as a therapeutic target for breast cancer treatment?
  Breast Cancer Research, 2011
• Inflammation and EMT: an alliance towards organ fibrosis and cancer progression
  EMBO Molecular Medicine, 2009
• EMT: When epithelial cells decide to become mesenchymal-like cells
  Journal of Clinical Investigation, 2009
• Chemoresistant Colorectal Cancer Cells, the Cancer Stem Cell Phenotype, and Increased Sensitivity to Insulin-like Growth Factor-I Receptor Inhibition
  Cancer Research, 2009
• γ-Secretase Inhibitors Abrogate Oxaliplatin-Induced Activation of the Notch-1 Signaling Pathway in Colon Cancer Cells Resulting in Enhanced Chemosensitivity
  Cancer Research, 2009
• Snail and Slug Promote Epithelial-Mesenchymal Transition through β-Catenin–T-Cell Factor-4-dependent Expression of Transforming Growth Factor-β3
  Molecular Biology of the Cell, 2008
• A Role for the Cleaved Cytoplasmic Domain of E-cadherin in the Nucleus
  Journal of Biological Chemistry, 2008
• The transcription factor Snail is a repressor of E-cadherin gene expression in epithelial tumour cells
  Nature Cell Biology, 2000
• Cleavage of Structural Proteins during the Assembly of the Head of Bacteriophage T4
  Nature, 1970