Glutathione S-transferase M1, T1, and P1 Polymorphisms and Parkinson's Disease

This publication has 17 references indexed in Scilit:

• An Association between Idiopathic Parkinson's Disease and Polymorphisms of Phase II Detoxification Enzymes: Glutathione S-Transferase M1 and Quinone Oxidoreductase 1 and 2
  Biochemical and Biophysical Research Communications, 2001
• Oxidative Damage Linked to Neurodegeneration by Selective α-Synuclein Nitration in Synucleinopathy Lesions
  Science, 2000
• Overexpression of human α-synuclein causes dopamine neuron death in rat primary culture and immortalized mesencephalon-derived cells
  Brain Research, 2000
• GSTM1 and mEPHX Polymorphisms in Parkinson's Disease and Age of Onset
  Biochemical and Biophysical Research Communications, 2000
• Determination of glutathione S-transferase μ and Θ polymorphisms in neurological disease
  Human & Experimental Toxicology, 1999
• ETIOLOGY AND PATHOGENESIS OF PARKINSON'S DISEASE
  Annual Review of Neuroscience, 1999
• Simultaneous characterization of glutathione S-transferase M1 and T1 polymorphisms by polymerase chain reaction in American whites and blacks
  Pharmacogenetics, 1996
• Oligonucleotides with fluorescent dyes at opposite ends provide a quenched probe system useful for detecting PCR product and nucleic acid hybridization.
  Genome Research, 1995
• Glutathion s‐transferase (class μ) phenotype in Parkinson's disease
  Movement Disorders, 1994
• Iron and Aluminum Increase in the Substantia Nigra of Patients with Parkinson's Disease: An X‐Ray Microanalysis
  Journal of Neurochemistry, 1991