Prolonged depletion of renal tubular thioredoxin following severe acute kidney injury is associated with transition to chronic kidney disease via G2/M cell cycle arrest
Open Access
- 3 February 2025
- journal article
- Published by Elsevier in Biochemical and Biophysical Research Communications
- Vol. 754, 151425
Abstract
No abstract availableKeywords
Funding Information
- Japan Agency for Medical Research and Development (JP22ym0126808)
- Japan Agency for Medical Research and Development (JP23ym0126808)
- Japan Agency for Medical Research and Development (JP19lm0203006)
- Ministry of Health, Labour and Welfare (6943371)
- Ministry of Health, Labour and Welfare (7270192)
- Ministry of Health, Labour and Welfare (7573284)
- Japan Society for the Promotion of Science (23K07695)
- Japan Society for the Promotion of Science (23K08228)
- Japan Society for the Promotion of Science (21K08252)
- Japan Society for the Promotion of Science (20H03696)
- University of Fukui
- Ministry of Education, Culture, Sports, Science and Technology
This publication has 45 references indexed in Scilit:
- Analytical and clinical validation of rapid chemiluminescence enzyme immunoassay for urinary thioredoxin, an oxidative stress-dependent early biomarker of acute kidney injuryClinica Chimica Acta, 2020
- AKI on CKD: heightened injury, suppressed repair, and the underlying mechanismsKidney International, 2017
- Quantified kidney echogenicity in mice with renal ischemia reperfusion injury: evaluation as a noninvasive biomarker of acute kidney injuryMedical Molecular Morphology, 2017
- Serum Levels of Thioredoxin Are Associated with Stroke Risk, Severity, and Lesion VolumesMolecular Neurobiology, 2014
- Polyamine depletion partially reduces the radiation-induced cell death via cell cycle delay mediated by thioredoxinCell Biology and Toxicology, 2006
- Thioredoxin overexpression in HT-1080 cells induced cellular senescence and sensitization to gamma radiationFEBS Letters, 2005
- Mitochondrial complex III is required for hypoxia-induced ROS production and cellular oxygen sensingCell Metabolism, 2005
- Increased oxidative stress with elevated serum thioredoxin level in patients with coronary spastic anginaClinical Cardiology, 2003
- Cell cycle regulation by the Cdc25 phosphatase family2000
- Early Embryonic Lethality Caused by Targeted Disruption of the Mouse Thioredoxin GeneDevelopmental Biology, 1996