Calvatia lilacina protein extract induces apoptosis through endoplasmic reticulum stress in human colon carcinoma cells
Authors:
- 31 August 2011
- journal article
- Published by Elsevier in Process Biochemistry
- Vol. 46 (8) , 1599-1606
Abstract
No abstract availableKeywords
This publication has 19 references indexed in Scilit:
- Reactive oxygen species-mediated endoplasmic reticulum stress and mitochondrial dysfunction contribute to cirsimaritin-induced apoptosis in human gallbladder carcinoma GBC-SD cellsCancer Letters, 2010
- Calvatia lilacinaProtein-Extract Induces Apoptosis through Glutathione Depletion in Human Colorectal Carcinoma CellsJournal of Agricultural and Food Chemistry, 2009
- Coupling mitochondrial dysfunction to endoplasmic reticulum stress response: A molecular mechanism leading to hepatic insulin resistanceCellular Signalling, 2009
- The Molecular Mechanism of Endoplasmic Reticulum Stress-Induced Apoptosis in PC-12 Neuronal Cells: The Protective Effect of Insulin-Like Growth Factor IEndocrinology, 2008
- Endoplasmic reticulum stress induced by oxidative stress in retinal pigment epithelial cellsGraefe's Archive for Clinical and Experimental Ophthalmology, 2008
- Aggravated Endoplasmic Reticulum Stress as a Basis for Enhanced Glioblastoma Cell Killing by Bortezomib in Combination with Celecoxib or Its Non-Coxib Analogue, 2,5-Dimethyl-CelecoxibCancer Research, 2008
- Calcium-activated endoplasmic reticulum stress as a major component of tumor cell death induced by 2,5-dimethyl-celecoxib, a non-coxib analogue of celecoxibMolecular Cancer Therapeutics, 2007
- Coping with stress: eIF2 kinases and translational controlBiochemical Society Transactions, 2006
- Regulation of apoptosis by endoplasmic reticulum pathwaysOncogene, 2003
- Cellular stress response and apoptosis in cancer therapyBlood, 2001