Amyloidosis and its management: Amyloid neuropathies
- 1 September 2016
- journal article
- review article
- Published by Elsevier in Current Problems in Cancer
- Vol. 40 (5-6) , 198-208
Abstract
No abstract availableThis publication has 48 references indexed in Scilit:
- Changes in pathological and biochemical findings of systemic tissue sites in familial amyloid polyneuropathy more than 10 years after liver transplantationJournal of Neurology, Neurosurgery & Psychiatry, 2013
- An amyotrophic lateral sclerosis-like syndrome revealing an amyloid polyneuropathy associated with a novel transthyretin mutationAmyloid, 2013
- Transthyretin Val30Met familial amyloid polyneuropathy: a considerably different clinical picture and natural course in endemic and non-endemic areasJournal of Neurology, Neurosurgery & Psychiatry, 2012
- Natural history of transthyretin Val30Met familial amyloid polyneuropathy: analysis of late-onset cases from non-endemic areasJournal of Neurology, Neurosurgery & Psychiatry, 2012
- Familial amyloid polyneuropathyThe Lancet Neurology, 2011
- Progression of transthyretin amyloid neuropathy after liver transplantationNeurology, 2010
- Primary amyloidosis presenting as upper limb multiple mononeuropathiesMuscle & Nerve, 2010
- Monoclonal gammopathy and neuropathyCurrent Opinion in Neurology, 2007
- Danish type gelsolin related amyloidosis: 654G-T mutation is associated with a disease pathogenetically and clinically similar to that caused by the 654G-A mutation (familial amyloidosis of the Finnish type)Journal of Clinical Pathology, 2000
- Familial amyloidosis, Finnish type: G654 → A mutation of the gelsolin gene in Finnish families and an unrelated American familyGenomics, 1992