Deactivation of 12(S)-HETE through (ω-1)-hydroxylation and β-oxidation in alternatively activated macrophages
Authors:
Open Access
- 1 April 2018
- journal article
- research article
- Published by Elsevier in Journal of Lipid Research
- Vol. 59 (4) , 615-624
Abstract
No abstract availableKeywords
Funding Information
- National Heart, Lung, and Blood Institute (HL-103673, HL-132908)
- Welch Foundation (I-0011)
This publication has 49 references indexed in Scilit:
- Mice lacking macrophage 12/15-lipoxygenase are resistant to experimental hypertensionAmerican Journal of Physiology-Heart and Circulatory Physiology, 2012
- Role of Tissue Macrophages in the Development of Critical Illness DiabetesShock, 2012
- Biochemistry and genetics of inherited disorders of peroxisomal fatty acid metabolismJournal of Lipid Research, 2010
- Structural characterization of monohydroxyeicosatetraenoic acids and dihydroxy- and trihydroxyeicosatrienoic acids by ESI-FTICRJournal of the American Society for Mass Spectrometry, 2008
- Monocyte and macrophage heterogeneityNature Reviews Immunology, 2005
- Inhibition of cytochrome P450 enzymes participating in p-nitrophenol hydroxylation by drugs known as CYP2E1 inhibitorsChemico-Biological Interactions, 2004
- CYP2U1, a Novel Human Thymus- and Brain-specific Cytochrome P450, Catalyzes ω- and (ω-1)-Hydroxylation of Fatty AcidsJournal of Biological Chemistry, 2004
- Increased Levels of 12(S)-HETE in Patients With Essential HypertensionHypertension, 2001
- Hydroxyeicosatetraenoic acid oxidation in chinese hamster ovary cells: a peroxisomal metabolic pathwayBiochimica et Biophysica Acta (BBA) - Lipids and Lipid Metabolism, 1991
- Thioridazine: a selective inhibitor of peroxisomal β‐oxidation in vivoFEBS Letters, 1985