Angiotensin II-induced pro-fibrotic effects require p38MAPK activity and transforming growth factor beta 1 expression in skeletal muscle cells

This publication has 45 references indexed in Scilit:

• Fibrotic response induced by angiotensin-II requires NAD(P)H oxidase-induced reactive oxygen species (ROS) in skeletal muscle cells
  Biochemical and Biophysical Research Communications, 2011
• Transient receptor potential melastatin 4 inhibition prevents lipopolysaccharide-induced endothelial cell death
  Cardiovascular Research, 2011
• Connective tissue growth factor induction by lysophosphatidic acid requires transactivation of transforming growth factor type β receptors and the JNK pathway
  Cellular Signalling, 2011
• A Novel Modulatory Mechanism of Transforming Growth Factor-β Signaling through Decorin and LRP-1
  Journal of Biological Chemistry, 2007
• Systemic infusion of angiotensin II exacerbates liver fibrosis in bile duct–ligated rats†
  Hepatology, 2005
• Liver fibrosis
  Journal of Clinical Investigation, 2005
• Connective tissue growth factor—a novel mediator of angiotensin II-stimulated cardiac fibroblast activation in heart failure in rats
  Journal of Molecular and Cellular Cardiology, 2004
• Lisofylline, a Novel Antiinflammatory Compound, Protects Mesangial Cells from Hyperglycemia- and Angiotensin II-Mediated Extracellular Matrix Deposition
  Endocrinology, 2003
• NADPH oxidase signal transduces angiotensin II in hepatic stellate cells and is critical in hepatic fibrosis
  Journal of Clinical Investigation, 2003
• Angiotensin II and Cardiac Fibrosis
  Trends in Cardiovascular Medicine, 1996