The adaptor molecule Disabled‐2 links the transforming growth factor β receptors to the Smad pathway
Authors:
Open Access
- 1 June 2001
- journal article
- Published by Springer Nature in The EMBO Journal
- Vol. 20 (11) , 2789-2801
Abstract
EMBO Press is an editorially independent publishing platform for the development of EMBO scientific publications.Keywords
This publication has 51 references indexed in Scilit:
- Disabled-2 inactivation is an early step in ovarian tumorigenicityOncogene, 1999
- Structure of a Numb PTB domain–peptide complex suggests a basis for diverse binding specificityNature Structural & Molecular Biology, 1998
- Heteromeric and Homomeric Transforming Growth Factor-β Receptors Show Distinct Signaling and Endocytic Responses in Epithelial CellsJournal of Biological Chemistry, 1998
- p96, a MAPK-related protein, is consistently downregulated during mouse mammary carcinogenesisOncogene, 1998
- DOC-2, a candidate tumor suppressor gene in human epithelial ovarian cancerOncogene, 1998
- Disabled-2 (Dab2) is an SH3 domain-binding partner of Grb2Oncogene, 1998
- Human Smad3 and Smad4 Are Sequence-Specific Transcription ActivatorsMolecular Cell, 1998
- Transforming growth factor β-induced phosphorylation of Smad3 is required for growth inhibition and transcriptional induction in epithelial cellsProceedings of the National Academy of Sciences, 1997
- Structure and ligand recognition of the phosphotyrosine binding domain of ShcNature, 1995
- Mechanism of activation of the TGF-β receptorNature, 1994