Host‐Pathogen Interactions Mediating Pain of Urinary Tract Infection
Authors:
Open Access
- 15 April 2010
- journal article
- Published by Oxford University Press (OUP) in The Journal of Infectious Diseases
- Vol. 201 (8) , 1240-1249
Abstract
Background. Pelvic pain is a major component of the morbidity associated with urinary tract infection (UTI), yet the molecular mechanisms underlyingKeywords
This publication has 48 references indexed in Scilit:
- Infectious Diseases Society of America Guidelines for the Diagnosis and Treatment of Asymptomatic Bacteriuria in AdultsClinical Infectious Diseases, 2005
- Uropathogenic Escherichia coli Potentiates Type 1 Pilus-Induced Apoptosis by Suppressing NF-κBInfection and Immunity, 2001
- Valency conversion in the type 1 fimbrial adhesin of Escherichia coliMolecular Microbiology, 2001
- Time course of LPS-induced gene expression in a mouse model of genitourinary inflammationPhysiological Genomics, 2001
- Type 1 fimbriae deliver an LPS‐ and TLR4‐dependent activation signal to CD14‐negative cellsMolecular Microbiology, 2001
- Induction of vanilloid receptor channel activity by protein kinase CNature, 2000
- Interleukin‐8 Receptor Knockout Mice Have Subepithelial Neutrophil Entrapment and Renal Scarring following Acute PyelonephritisThe Journal of Infectious Diseases, 2000
- Determination of mouse bladder inflammatory response to E. coli lipopolysaccharideUrological Research, 2000
- Quantification of Bacterial Lipopolysaccharides by the Purpald Assay: Measuring Formaldehyde Generated from 2-keto-3-deoxyoctonate and Heptose at the Inner Core by Periodate OxidationAnalytical Biochemistry, 1999
- Defective LPS Signaling in C3H/HeJ and C57BL/10ScCr Mice: Mutations in Tlr4 GeneScience, 1998