Activation of the neuronal c-Abl tyrosine kinase by amyloid-β-peptide and reactive oxygen species
Authors:
- Alejandra R. AlvarezAlejandra R. Alvarez
- Pablo C. SandovalPSPablo C. Sandoval
- Nancy R. LealNLNancy R. Leal
- Paula U. CastroPCPaula U. Castro
- Kenneth S. KosikKKKenneth S. Kosik
- 1 November 2004
- journal article
- Published by Elsevier in Neurobiology of Disease
- Vol. 17 (2) , 326-336
Abstract
No abstract availableKeywords
This publication has 45 references indexed in Scilit:
- Amyloid-β: a chameleon walking in two worlds: a review of the trophic and toxic properties of amyloid-βBrain Research Reviews, 2003
- c-Abl Tyrosine Kinase Selectively Regulates p73 Nuclear Matrix AssociationJournal of Biological Chemistry, 2003
- Glivec (STI571, imatinib), a rationally developed, targeted anticancer drugNature Reviews Drug Discovery, 2002
- c-Abl Has High Intrinsic Tyrosine Kinase Activity That Is Stimulated by Mutation of the Src Homology 3 Domain and by Autophosphorylation at Two Distinct Regulatory TyrosinesJournal of Biological Chemistry, 2000
- Inhibition of tau phosphorylating protein kinase cdk5 prevents β‐amyloid‐induced neuronal deathFEBS Letters, 1999
- The tyrosine kinase c-Abl regulates p73 in apoptotic response to cisplatin-induced DNA damageNature, 1999
- Interaction of c-Abl and p73α and their collaboration to induce apoptosisNature, 1999
- Stable Complexes Involving Acetylcholinesterase and Amyloid-β Peptide Change the Biochemical Properties of the Enzyme and Increase the Neurotoxicity of Alzheimer’s FibrilsJournal of Neuroscience, 1998
- A rapid micrqpreparation technique for extraction of DNA-binding proteins from limiting numbers of mammalian cellsNucleic Acids Research, 1991
- MAP2 is localized to the dendrites of hippocampal neurons which develop in cultureDevelopmental Brain Research, 1984