Effect of agalsidase alfa replacement therapy on fabry disease—related hypertrophic cardiomyopathy: A 12- to 36-month, retrospective, blinded echocardiographic pooled analysis
Authors:
- Christoph KampmannCKChristoph Kampmann
- Aleš LinhartALAleš Linhart
- Richard B. DevereuxRDRichard B. Devereux
- Raphael SchiffmannRSRaphael Schiffmann
- 1 September 2009
- journal article
- research article
- Published by Elsevier in Clinical Therapeutics
- Vol. 31 (9) , 1966-1976
Abstract
No abstract availableKeywords
This publication has 44 references indexed in Scilit:
- Treatment of Anderson–Fabry diseaseHeart, 2008
- Impact of Enzyme Replacement Therapy on Cardiac Morphology and Function and Late Enhancement in Fabry’s CardiomyopathyThe American Journal of Cardiology, 2006
- The effect of 12‐month enzyme replacement therapy on myocardial perfusion in patients with Fabry diseaseJournal of Inherited Metabolic Disease, 2006
- Prevalence and Clinical Significance of Cardiac Arrhythmia in Anderson-Fabry DiseaseThe American Journal of Cardiology, 2005
- Coronary microvascular dysfunction in male patients with Anderson-Fabry disease and the effect of treatment with α galactosidase AHeart, 2005
- The natural history of left ventricular systolic function in Anderson-Fabry diseaseHeart, 2005
- Electrophysiologic findings in Fabry's disease with a short PR intervalThe American Journal of Cardiology, 1994
- Detection of left ventricular hypertrophy by M-mode echocardiography. Anatomic validation, standardization, and comparison to other methods.Hypertension, 1987
- Echocardiographic abnormalities and disease severity in Fabry's diseaseJACC, 1986
- Recommendations regarding quantitation in M-mode echocardiography: results of a survey of echocardiographic measurements.Circulation, 1978