Leptin, Endothelin, NADPH Oxidase, and Heart Failure

This publication has 10 references indexed in Scilit:

• Leptin Regulates Cardiomyocyte Contractile Function Through Endothelin-1 Receptor–NADPH Oxidase Pathway
  Hypertension, 2006
• Impaired cardiac contractile function in ventricular myocytes from leptin-deficient ob/ob obese mice
  Journal of Endocrinology, 2006
• Leptin repletion restores depressed β‐adrenergic contractility in ob/ob mice independently of cardiac hypertrophy
  The Journal of Physiology, 2005
• Endothelin‐1 enhances oxidative stress, cell proliferation and reduces apoptosis in human umbilical vein endothelial cells: role of ET_B receptor, NADPH oxidase and caveolin‐1
  British Journal of Pharmacology, 2005
• Endothelium-Restricted Overexpression of Human Endothelin-1 Causes Vascular Remodeling and Endothelial Dysfunction
  Circulation, 2004
• Novel NAD(P)H oxidases in the cardiovascular system: Table 1
  Heart, 2004
• Conditional Cardiac Overexpression of Endothelin-1 Induces Inflammation and Dilated Cardiomyopathy in Mice
  Circulation, 2004
• Abrogated Leptin-Induced Cardiac Contractile Response in Ventricular Myocytes Under Spontaneous Hypertension
  Hypertension, 2002
• Endothelin-1 Induces NAD(P)H Oxidase in Human Endothelial Cells
  Biochemical and Biophysical Research Communications, 2000
• Hyperleptinaemia does not correlate with plasma catecholamine levels in chronic heart failure
  1999