Triclocarban induces lipid droplet accumulation and oxidative stress responses by inhibiting mitochondrial fatty acid oxidation in HepaRG cells
Open Access
- 15 April 2024
- journal article
- research article
- Published by Elsevier in Toxicology Letters
- Vol. 396, 11-18
Abstract
No abstract availableKeywords
This publication has 33 references indexed in Scilit:
- Association of triclosan and triclocarban in urine with obesity risk in Chinese school childrenEnvironment International, 2021
- Fate and biodegradation characteristics of triclocarban in wastewater treatment plants and sewage sludge composting processes and risk assessment after entering the ecological environmentJournal of Hazardous Materials, 2021
- In vitro effects of Triclocarban on adipogenesis in murine preadipocyte and human hepatocyteJournal of Hazardous Materials, 2020
- Triclocarban Disrupts the Epigenetic Status of Neuronal Cells and Induces AHR/CAR-Mediated ApoptosisMolecular Neurobiology, 2018
- HuH-7 reference genome profile: complex karyotype composed of massive loss of heterozygosityHuman Cell, 2018
- Cell culture models of fatty acid overload: Problems and solutionsBiochimica et Biophysica Acta (BBA) - Molecular and Cell Biology of Lipids, 2018
- Assessment of Fatty Acid Beta Oxidation in Cells and Isolated MitochondriaCurrent Protocols in Toxicology, 2014
- Whole blood is the sample matrix of choice for monitoring systemic triclocarban levelsChemosphere, 2012
- Inhibitors of fatty acid synthesis as antimicrobial chemotherapeuticsApplied Microbiology and Biotechnology, 2002
- Inhibition of mitochondrial beta-oxidation as a mechanism of hepatotoxicityPharmacology & Therapeutics, 1995