Activation of the Transcription Factor NF-κB in the Nucleus Trigeminalis Caudalis in an Animal Model of Migraine
Authors:
- Rosaria GrecoRGRosaria Greco
- Cristina TassorelliCTCristina Tassorelli
- Donata CappellettiDCDonata Cappelletti
- Giorgio SandriniGSGiorgio Sandrini
- Giuseppe NappiGNGiuseppe Nappi
- 1 October 2005
- journal article
- Published by Elsevier in NeuroToxicology
- Vol. 26 (5) , 795-800
Abstract
No abstract availableKeywords
This publication has 27 references indexed in Scilit:
- Parthenolide is the Component of Tanacetum Parthenium that Inhibits Nitroglycerin-Induced Fos Activation: Studies in an Animal Model of MigraineCephalalgia, 2005
- Biphasic Response to Nitric Oxide of Spinal Trigeminal Neurons With Meningeal Input in Rat–Possible Implications for the Pathophysiology of HeadachesJournal of Neurophysiology, 2004
- Spinal NF‐kB activation induces COX‐2 upregulation and contributes to inflammatory pain hypersensitivityEuropean Journal of Neuroscience, 2004
- Nitroglycerin-Induced nNOS Increase in Rat Trigeminal Nucleus Caudalis is Inhibited by Systemic Administration of Lysine Acetylsalicylate but not of SumatriptanCephalalgia, 2004
- Nitroglycerin enhances cGMP expression in specific neuronal and cerebrovascular structures of the rat brainJournal of Chemical Neuroanatomy, 2004
- Central Components of the Analgesic/Antihyperalgesic Effect of NimesulideDrugs, 2003
- NF-κB decoy suppresses cytokine expression and thermal hyperalgesia in a rat neuropathic pain modelNeuroReport, 2001
- The effects on the central nervous system of nitroglycerin—putative mechanisms and mediatorsProgress in Neurobiology, 1999
- Nitric oxide supersensitivity: a possible molecular mechanism of migraine painNeuroReport, 1993
- Tissue levels of glyceryl trinitrate and cGMP after in vivo administration in rat, and the effect of tolerance developmentCanadian Journal of Physiology and Pharmacology, 1991