DOT1L-mediated H3K79me1 transcriptional activation of Acp5 aggravates inflammatory responses following diabetic vascular injury
- 2 November 2025
- journal article
- Published by Elsevier in Microvascular Research
- Vol. 163, 104882
Abstract
No abstract availableKeywords
Funding Information
- National Natural Science Foundation of China (82170418)
- National Natural Science Foundation of China (82471616)
- National Natural Science Foundation of China (82271618)
- Natural Science Foundation of Hubei Province (2022CFA015)
- Science and Technology Program of Hubei Province (2025CCB016)
- Hebei Provincial Key Research Projects
- Hubei Three Gorges Laboratory
This publication has 31 references indexed in Scilit:
- Neointima abating and endothelium preserving — An adventitia-localized nanoformulation to inhibit the epigenetic writer DOT1LBiomaterials, 2023
- DOT1L regulates lipid biosynthesis and inflammatory responses in macrophages and promotes atherosclerotic plaque stabilityCell Reports, 2022
- Nullifying epigenetic writer DOT1L attenuates neointimal hyperplasiaAtherosclerosis, 2020
- Usefulness of Routine Fractional Flow Reserve for Clinical Management of Coronary Artery Disease in Patients With DiabetesJAMA Cardiology, 2020
- MicroRNA-24 inhibits the oxidative stress induced by vascular injury by activating the Nrf2/Ho-1 signaling pathwayAtherosclerosis, 2019
- Outcomes Among Diabetic Patients Undergoing Percutaneous Coronary Intervention With Contemporary Drug-Eluting StentsJACC: Cardiovascular Interventions, 2018
- Diabetes propels the risk for cardiovascular disease: sweet monocytes becoming aggressive?Cellular and Molecular Life Sciences, 2016
- Tartrate‐Resistant Acid Phosphatase Deficiency in the Predisposition to Systemic Lupus ErythematosusArthritis & Rheumatology, 2016
- Analysis of β-catenin alterations in colon tumors: a novel exon 3 mutationTumor Biology, 2010
- Methylation of H3-Lysine 79 Is Mediated by a New Family of HMTases without a SET DomainCurrent Biology, 2002