HDAC inhibition helps post-MI healing by modulating macrophage polarization
Authors:
- 1 June 2018
- journal article
- research article
- Published by Elsevier in Journal of Molecular and Cellular Cardiology
- Vol. 119, 51-63
Abstract
No abstract availableKeywords
Funding Information
- United States Department of Veterans Affairs Merit Review (BX002327)
- NIH (TR001450)
- NCATS (T32HL07260)
This publication has 44 references indexed in Scilit:
- Monocyte-Directed RNAi Targeting CCR2 Improves Infarct Healing in Atherosclerosis-Prone MiceCirculation, 2013
- Novel therapeutic approaches to post-infarction remodellingCardiovascular Research, 2012
- Temporal and Spatial Expression of Matrix Metalloproteinases and Tissue Inhibitors of Metalloproteinases Following Myocardial InfarctionCardiovascular Therapeutics, 2012
- Histone deacetylase 3 is an epigenomic brake in macrophage alternative activationGenes & Development, 2011
- Macrophage polarization in the maculae of age‐related macular degeneration: A pilot studyPathology International, 2011
- Modulation of cardiac macrophages by phosphatidylserine-presenting liposomes improves infarct repairProceedings of the National Academy of Sciences, 2011
- Human Gingiva-Derived Mesenchymal Stem Cells Elicit Polarization of M2 Macrophages and Enhance Cutaneous Wound HealingStem Cells, 2010
- Impaired Infarct Healing in Atherosclerotic Mice With Ly-6ChiMonocytosisJACC, 2010
- Identification of Two Distinct Macrophage Subsets with Divergent Effects Causing either Neurotoxicity or Regeneration in the Injured Mouse Spinal CordThe Journal of Neuroscience, 2009
- Mechanisms of postinfarct left ventricular remodelingDrug Discovery Today: Disease Mechanisms, 2007