Decreased potassium-stimulated release of [3H]d-aspartate from hippocampal slices distinguishes encephalopathy related to acute liver failure from that induced by simple hyperammonemia
Authors:
- Renata HaugvicovaRHRenata Haugvicova
- Jan AlbrechtJAJan Albrecht
- 1 December 1991
- journal article
- research article
- Published by Elsevier in Brain Research
- Vol. 567 (1) , 165-168
Abstract
No abstract availableKeywords
This publication has 22 references indexed in Scilit:
- Effect of Portacaval Anastomosis on Electrically Stimulated Release of Glutamate from Rat Hippocampal SlicesJournal of Neurochemistry, 1991
- Neurochemical and electrophysiological studies on the inhibitory effect of ammonium ions on synaptic transmission in slices of rat hippocampus: Evidence for a postsynaptic actionNeuroscience, 1990
- GABAA Receptor Complex in an Experimental Model of Hepatic Encephalopathy: Evidence for Elevated Levels of an Endogenous Benzodiazepine Receptor LigandJournal of Neurochemistry, 1989
- l-Glutamate stimulates the efflux of newly taken up glutamine from astroglia but not from synaptosomes of the ratNeuropharmacology, 1989
- Ammonia: Key factor in the pathogenesis of hepatic encephalopathyNeurochemical Pathology, 1987
- Brain carbonic anhydrase activity in rats in experimental hepatogenic encephalopathyNeuroscience Letters, 1984
- Changes in Glutamate Receptors on Synaptic Membranes Associated with Hepatic Encephalopathy or Hyperammonemia in the RabbitHepatology, 1984
- Characterization of uptake and release processes ford- andl-aspartate in primary cultures of astrocytes and cerebellar granule cellsNeurochemical Research, 1983
- AMMONIUM ION INHIBITION OF EVOKED RELEASE OF ENDOGENOUS GLUTAMATE FROM HIPPOCAMPAL SLICESJournal of Neurochemistry, 1979
- The HippocampusPhysiological Reviews, 1964