NONALCOHOLIC CHRONIC HEPATITIS IN ALCOHOLIC

Authors:
  • SGS J Goldberg
    S J Goldberg
  • CMC L Mendenhall
    C L Mendenhall
  • ACA M Connell
    A M Connell
  • ACA Chedid
    A Chedid
Google Scholar
  • 1 January 1977
    • journal article
    • research article
    • Vol. 72  (4) , 598-604
    • PMID:838212
Abstract
Ten alcoholic patients with biopsy proved chronic active or chronic persistent hepatitis were observed. In each patient, the responsible etiological agent appeared to be ethanol. Laboratory abnormalities could be distinguished statistically from those in a group of 121 patients with alcoholic hepatitis by their higher SGPT [serum glutamic pyruvic transaminase] (262 .+-. 139 vs. 62 .+-. 7 U[units]/ml, P < 0.01), lower ratio of SGOT[serum glutamic oxalacetic transaminase]:SGPT (1.96 .+-. 0.34 vs. 4.71 .+-. 0.40, P < 0.01) and lower white blood cell count (5833 .+-. 763 vs. 10,370 .+-. 742, P L 0.01). The overlap between the groups was sufficiently large that without histological confirmation the correct diagnosis was in doubt for any given patient.

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