Cytokine imbalance at materno-embryonic interface as a potential immune mechanism for recurrent pregnancy loss
- 1 January 2021
- journal article
- research article
- Published by Elsevier in International Immunopharmacology
- Vol. 90, 107118
Abstract
No abstract availableKeywords
This publication has 276 references indexed in Scilit:
- The IL-10/STAT3-mediated anti-inflammatory response: recent developments and future challengesBriefings in Functional Genomics, 2013
- Inducing Tolerance to PregnancyNew England Journal of Medicine, 2012
- Pregnancy-associated diseases are characterized by the composition of the systemic regulatory T cell (Treg) pool with distinct subsets of TregsClinical and Experimental Immunology, 2011
- Natural killer cell-triggered vascular transformation: maternal care before birth?Cellular & Molecular Immunology, 2010
- REVIEW ARTICLE: Interleukin‐10: A Multi‐Faceted Agent of PregnancyAmerican Journal of Reproductive Immunology, 2010
- Plasticity of T‐cell phenotype and function: the T helper type 17 exampleImmunology, 2010
- Translational Mini-Review Series on Th17 Cells: Function and regulation of human T helper 17 cells in health and diseaseClinical and Experimental Immunology, 2009
- Prokineticin-1 (PROK1) modulates interleukin (IL)-11 expression via prokineticin receptor 1 (PROKR1) and the calcineurin/NFAT signalling pathwayMolecular Human Reproduction, 2009
- Interferon Gamma in Successful Pregnancies1Biology of Reproduction, 2009
- Maternal heparin-binding-EGF deficiency limits pregnancy success in miceProceedings of the National Academy of Sciences, 2007