Involvement of Akt, Ras and cell cycle regulators in the potential development of endometrial hyperplasia in women with polycystic ovarian syndrome
- 1 October 2009
- journal article
- research article
- Published by Elsevier in Gynecologic Oncology
- Vol. 115 (1) , 102-107
Abstract
No abstract availableKeywords
Funding Information
- National Cancer Institute
- Fondo Nacional de Desarrollo Científico y Tecnológico (K22CA121991, PRE 070/2003)
This publication has 50 references indexed in Scilit:
- Deregulation of tissue homeostasis in endometria from patients with polycystic ovarian syndrome with and without endometrial hyperplasiaGynecologic Oncology, 2007
- Expression of survivin, PTEN and p27 in normal, hyperplastic, and carcinomatous endometriumInternational Journal of Gynecological Cancer, 2006
- Tumorigenesis of K-ras mutation in human endometrial carcinoma via upregulation of estrogen receptorGynecologic Oncology, 2006
- Prevalence and clinical predictors of endometrial hyperplasiain anovulatory women presenting with amenorrheaGynecological Endocrinology, 2006
- Endometrial cancer in young, normal-weight womenGynecologic Oncology, 2005
- Estrogen-induced proliferation of normal endometrial glandular cells is initiated by transcriptional activation of cyclin D1 via binding of c-Jun to an AP-1 sequenceOncogene, 2004
- RAS and RHO GTPases in G1-phase cell-cycle regulationNature Reviews Molecular Cell Biology, 2004
- Are p27 and p21 Cytoplasmic Oncoproteins?Cell Cycle, 2002
- Role of nitric oxide and bcl-2 family genes in the regulation of human endometrial apoptosisFertility and Sterility, 2002
- Ultrasound and menstrual history in predicting endometrial hyperplasia in polycystic ovary syndromeObstetrics & Gynecology, 2001