Atypical Cross Talk Between Mentalizing and Mirror Neuron Networks in Autism Spectrum Disorder

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Abstract
Humans are an inherently social species. Our survival and success depend on our ability to navigate and thrive in complex social situations. This core ability is commonly impaired in autism spectrum disorder (ASD), a neurodevelopmental disorder affecting as many as 1 in 88 children.1 Despite the highly heterogeneous symptom manifestation, impairments in social functioning, including diminished social responsiveness, difficulty relating to others, and recognizing others’ emotions and intentions, are defining features of ASD.2 These social deficits are considered the most universal and specific characteristics of ASD,3 both defining and distinguishing it from other developmental disorders.4 Yet, the neural mechanisms underlying social impairments remain largely undetermined, despite attracting a great deal of research.

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