GABAB Receptor Signaling in the Mesolimbic System Suppresses Binge-like Consumption of a High-Fat Diet

This publication has 42 references indexed in Scilit:

• Lack of GPR88 enhances medium spiny neuron activity and alters motor- and cue-dependent behaviors
  Nature Neuroscience, 2012
• Dopamine D2 receptors in addiction-like reward dysfunction and compulsive eating in obese rats
  Nature Neuroscience, 2010
• Human genetics illuminates the paths to metabolic disease
  Nature, 2009
• Baclofen suppresses binge eating of pure fat but not a sugar-rich or sweet–fat diet
  Behavioural Pharmacology, 2009
• Striatal GPR88 expression is confined to the whole projection neuron population and is regulated by dopaminergic and glutamatergic afferents
  European Journal of Neuroscience, 2009
• Baclofen, raclopride, and naltrexone differentially affect intake of fat/sucrose mixtures under limited access conditions
  Pharmacology Biochemistry and Behavior, 2009
• Activation of mesolimbic dopamine neurons during novel and daily limited access to palatable food is blocked by the opioid antagonist LY255582
  American Journal of Physiology-Regulatory, Integrative and Comparative Physiology, 2008
• Baclofen, raclopride, and naltrexone differentially reduce solid fat emulsion intake under limited access conditions
  Pharmacology Biochemistry and Behavior, 2008
• Peripherally administered baclofen reduced food intake and body weight in db/db as well as diet‐induced obese mice
  FEBS Letters, 2007
• Sham feeding corn oil increases accumbens dopamine in the rat
  American Journal of Physiology-Regulatory, Integrative and Comparative Physiology, 2006