Isothiocyanates Reduce Mercury Accumulation via an Nrf2-Dependent Mechanism during Exposure of Mice to Methylmercury
Open Access
- 1 August 2011
- journal article
- Published by American Chemical Society (ACS) in Environmental Health Perspectives
- Vol. 119 (8) , 1117-1122
Abstract
A peer-reviewed open access journal published by the National Institute of Environmental Health SciencesKeywords
This publication has 46 references indexed in Scilit:
- Adverse Effects of Methylmercury: Environmental Health Research ImplicationsEnvironmental Health Perspectives, 2010
- Methylmercury Induces Acute Oxidative Stress, Altering Nrf2 Protein Level in Primary Microglial CellsToxicological Sciences, 2010
- Targeting NRF2 signaling for cancer chemopreventionToxicology and Applied Pharmacology, 2010
- A food‐derived synergist of NGF signaling: identification of protein tyrosine phosphatase 1B as a key regulator of NGF receptor‐initiated signal transductionJournal of Neurochemistry, 2008
- Methylmercury Toxicity and Nrf2-dependent Detoxification in AstrocytesToxicological Sciences, 2008
- Associations of maternal long-chain polyunsaturated fatty acids, methyl mercury, and infant development in the Seychelles Child Development Nutrition StudyNeuroToxicology, 2008
- Cytoprotective role of Nrf2/Keap1 system in methylmercury toxicityBiochemical and Biophysical Research Communications, 2007
- Oxidative and electrophilic stress induces multidrug resistance–associated protein transporters via the nuclear factor-E2–related factor-2 transcriptional pathwayHepatology, 2007
- Sulforaphane, an activator of Nrf2, suppresses cellular accumulation of arsenic and its cytotoxicity in primary mouse hepatocytesFEBS Letters, 2006
- Inhibitory Effect of Selenium on Biliary Secretion of Methyl Mercury in RatsBiochemical and Biophysical Research Communications, 1997