Fuchs' endothelial corneal dystrophy: pathology and treatment outcome
Author:
- 7 November 2016
- journal article
- Published by Wiley in Acta Ophthalmologica
- Vol. 94 (A104) , 1-27
Abstract
No abstract availableThis publication has 101 references indexed in Scilit:
- Decline in DJ-1 and Decreased Nuclear Translocation of Nrf2 in Fuchs Endothelial Corneal DystrophyInvestigative Ophthalmology & Visual Science, 2012
- The genetics of Fuchs’ corneal dystrophyExpert Review of Ophthalmology, 2012
- Visual Performance with Wave Aberration Correction after Penetrating, Deep Anterior Lamellar, or Endothelial KeratoplastyInvestigative Ophthalmology & Visual Science, 2012
- The impact of the unfolded protein response on human diseaseJournal of Cell Biology, 2012
- Mutations in LOXHD1, a Recessive-Deafness Locus, Cause Dominant Late-Onset Fuchs Corneal DystrophyThe American Journal of Human Genetics, 2012
- A cellular model for the investigation of Fuchs' Endothelial Corneal DystrophyExperimental Eye Research, 2011
- An alpha 2 collagen VIII transgenic knock-in mouse model of Fuchs endothelial corneal dystrophy shows early endothelial cell unfolded protein response and apoptosisHuman Molecular Genetics, 2011
- Missense mutations in the sodium borate cotransporter SLC4A11 cause late-onset Fuchs corneal dystrophyaHuman Mutation, 2010
- Unfolded Protein Response in Fuchs Endothelial Corneal Dystrophy: A Unifying Pathogenic Pathway?American Journal of Ophthalmology, 2010
- Missense Mutations in TCF8 Cause Late-Onset Fuchs Corneal Dystrophy and Interact with FCD4 on Chromosome 9pThe American Journal of Human Genetics, 2010