Aged Mice Exhibit Severe Exacerbations of Dry Eye Disease with an Amplified Memory Th17 Cell Response
- 11 April 2020
- journal article
- research article
- Published by Elsevier in The American Journal of Pathology
- Vol. 190 (7) , 1474-1482
Abstract
No abstract availableKeywords
Funding Information
- National Institutes of Health
This publication has 65 references indexed in Scilit:
- Ocular surface immunity: Homeostatic mechanisms and their disruption in dry eye diseaseProgress in Retinal and Eye Research, 2012
- Dry Eye DiseaseArchives of Ophthalmology (1950), 2012
- Autoimmunity at the ocular surface: pathogenesis and regulationMucosal Immunology, 2010
- Role of Th17 cells in the immunopathogenesis of dry eye diseaseMucosal Immunology, 2009
- IL-17 disrupts corneal barrier following desiccating stressMucosal Immunology, 2009
- TH17 cells contribute to uveitis and scleritis and are expanded by IL-2 and inhibited by IL-27/STAT1Nature Medicine, 2007
- Antiviral CD4+ memory T cells are IL-15 dependentJournal of Experimental Medicine, 2007
- Impact of Dry Eye Syndrome on Vision-Related Quality of LifeAmerican Journal of Ophthalmology, 2007
- The IL-15/IL-15Rα on cell surfaces enables sustained IL-15 activity and contributes to the long survival of CD8 memory T cellsProceedings of the National Academy of Sciences, 2007
- Aging of the Immune System: How Much Can the Adaptive Immune System Adapt?Immunity, 2006