Functional roles of p120ctn family of proteins in central neurons
- 1 September 2017
- journal article
- review article
- Published by Elsevier in Seminars in Cell & Developmental Biology
- Vol. 69, 70-82
Abstract
No abstract availableKeywords
Funding Information
- Munroe-Meyer Institute (5P20GM103471-10)
- Alzheimer’s Association (EPS-1004094)
- National Institute of General Medical Sciences of the National Institutes of Health (1R03MH110726-01)
- Nebraska EPSCoR
- RO3 from the National Institutes of Health
This publication has 117 references indexed in Scilit:
- Synaptic amplification by dendritic spines enhances input cooperativityNature, 2012
- Systematic and Quantitative Assessment of the Ubiquitin-Modified ProteomeMolecular Cell, 2011
- Cadherin-9 Regulates Synapse-Specific Differentiation in the Developing HippocampusNeuron, 2011
- A Tissue-Specific Atlas of Mouse Protein Phosphorylation and ExpressionCell, 2010
- δ-Catenin Is Required for the Maintenance of Neural Structure and Function in Mature Cortex In VivoNeuron, 2009
- E-Cadherin negatively modulates δ-catenin-induced morphological changes and RhoA activity reduction by competing with p190RhoGEF for δ-cateninBiochemical and Biophysical Research Communications, 2008
- Recurrent CNVs Disrupt Three Candidate Genes in Schizophrenia PatientsThe American Journal of Human Genetics, 2008
- Identification of E2F1 as a positive transcriptional regulator for δ-cateninBiochemical and Biophysical Research Communications, 2008
- Presenilin-1 inhibits δ-catenin-induced cellular branching and promotes δ-catenin processing and turnoverBiochemical and Biophysical Research Communications, 2006
- p120 Catenin Regulates Dendritic Spine and Synapse Development through Rho-Family GTPases and CadherinsNeuron, 2006