1,3-Dichloro-2-propanol induced hyperlipidemia in C57BL/6J mice via AMPK signaling pathway
Authors:
- 1 February 2014
- journal article
- research article
- Published by Elsevier in Food and Chemical Toxicology
- Vol. 64, 403-409
Abstract
No abstract availableKeywords
This publication has 35 references indexed in Scilit:
- Obesity and hyperlipidemia are risk factors for early diabetic neuropathyJournal of Diabetes and its Complications, 2013
- Increased risk of hyperlipidemia in patients with major depressive disorder: A population-based studyJournal of Psychosomatic Research, 2013
- AMPK regulation of fatty acid metabolism and mitochondrial biogenesis: Implications for obesityMolecular and Cellular Endocrinology, 2013
- Compliance with the Adult Treatment Panel III guidelines for hyperlipidemia in a resident-run ambulatory clinic: A retrospective data analysisJournal of Clinical Lipidology, 2013
- 1,3-Dichloro-2-propanol induces apoptosis via both calcium and ROS in mouse melanoma cellsBiotechnology Letters, 2009
- Time-Dependent Association Between Metabolic Syndrome and Risk of CKD in Korean Men Without Hypertension or DiabetesAmerican Journal of Kidney Diseases, 2009
- Structural basis for AMP binding to mammalian AMP-activated protein kinaseNature, 2007
- Dose-Dependent Effects of Dichloropropanol on Liver Histology and Lipid Peroxidation in Rats.Industrial Health, 1998
- Characterization of the AMP-activated Protein Kinase Kinase from Rat Liver and Identification of Threonine 172 as the Major Site at Which It Phosphorylates AMP-activated Protein KinaseJournal of Biological Chemistry, 1996
- Negative interactions between phosphorylation of acetyl‐CoA carboxylase by the cyclic AMP‐dependent and AMP‐activated protein kinasesFEBS Letters, 1988