Arginine-vasopressin V1a receptor inhibition improves neurologic outcomes following an intracerebral hemorrhagic brain injury
Authors:
- 1 March 2011
- journal article
- Published by Elsevier in Neurochemistry International
- Vol. 58 (4) , 542-548
Abstract
No abstract availableKeywords
This publication has 39 references indexed in Scilit:
- Do Current Animal Models of Intracerebral Hemorrhage Mirror the Human Pathology?Translational Stroke Research, 2010
- Arginine-Vasopressin V1 but not V2 Receptor Antagonism Modulates Infarct Volume, Brain Water Content, and Aquaporin-4 Expression Following Experimental StrokeNeurocritical Care, 2009
- Temporal changes in expression of aquaporin3, -4, -5 and -8 in rat brains after permanent focal cerebral ischemiaBrain Research, 2009
- Hypothermia Reduces Cytotoxic Edema and Metabolic Alterations during the Acute Phase of Massive SAH: A Diffusion-Weighted Imaging and Spectroscopy Study in RatsJournal of Neurotrauma, 2008
- Physiology and pathophysiology of the vasopressin-regulated renal water reabsorptionPflügers Archiv - European Journal of Physiology, 2008
- The modulation of aquaporin-4 by using PKC-activator (phorbol myristate acetate) and V1a receptor antagonist (SR49059) following middle cerebral artery occlusion/reperfusion in the rat2008
- Vasopressin serum levels and disorders of sodium and water balance in patients with severe brain injuryArquivos de Neuro-Psiquiatria, 2007
- NC-1900, an arginine–vasopressin analogue, ameliorates social behavior deficits and hyperlocomotion in MK-801-treated rats: Therapeutic implications for schizophreniaBrain Research, 2005
- The neurobiology of glia in the context of water and ion homeostasisNeuroscience, 2004
- Intracerebral hemorrhageCritical Care Medicine, 1993