Heparin attenuates lipopolysaccharide-induced acute lung injury by inhibiting nitric oxide synthase and TGF-β/Smad signaling pathway
Authors:
- Renyu DingRDRenyu Ding
- Xin AnXAXin An
- Xin LiXLXin Li
- Song ChenSCSong Chen
- Xiaochun MaXMXiaochun Ma
- 28 October 2011
- journal article
- research article
- Published by Elsevier in Thrombosis Research
- Vol. 129 (4) , 479-485
Abstract
No abstract availableKeywords
This publication has 34 references indexed in Scilit:
- The Acute Respiratory Distress Syndrome: Pathogenesis and TreatmentAnnual Review of Pathology: Mechanisms of Disease, 2011
- Incidence and Outcomes of Pediatric Acute Lung InjuryPediatrics, 2009
- Recent trends in acute lung injury mortality: 1996–2005*Critical Care Medicine, 2009
- Therapeutic Effects and Anti‐inflammatory Mechanisms of Heparin on Acute Lung Injury in RabbitsAcademic Emergency Medicine, 2008
- Acute Lung Injury and the Acute Respiratory Distress SyndromeAmerican Journal of Respiratory Cell and Molecular Biology, 2005
- Novel drug development opportunities for heparinNature Reviews Drug Discovery, 2002
- The Acute Respiratory Distress SyndromeNew England Journal of Medicine, 2000
- Coagulation Abnormalities in Acute Lung Injury and SepsisAmerican Journal of Respiratory Cell and Molecular Biology, 2000
- Function of pulmonary M2 muscarinic receptors in antigen-challenged guinea pigs is restored by heparin and poly-L-glutamate.Journal of Clinical Investigation, 1992
- Acidic polyamino acids inhibit human eosinophil granule major basic protein toxicity. Evidence of a functional role for ProMBP.Journal of Clinical Investigation, 1991