Oncogenic potential of PIK3CD in glioblastoma is exerted through cytoskeletal proteins PAK3 and PLEK2
- 18 July 2022
- journal article
- research article
- Published by Elsevier in Laboratory Investigation
- Vol. 102 (12) , 1314-1322
Abstract
No abstract availableKeywords
Funding Information
- National Natural Science Foundation of China (81402064, 81402064)
This publication has 43 references indexed in Scilit:
- Deciphering the link between PI3K and PAK: An opportunity to target key pathways in pancreatic cancer?Oncotarget, 2016
- Activated-PAK4 predicts worse prognosis in breast cancer and promotes tumorigenesis through activation of PI3K/AKT signalingOncotarget, 2016
- P21 activated kinasesSmall GTPases, 2014
- Erlotinib resistance in EGFR-amplified glioblastoma cells is associated with upregulation of EGFRvIII and PI3Kp110Neuro-Oncology, 2013
- The catalytic phosphoinositol 3-kinase isoform p110δ is required for glioma cell migration and invasionEuropean Journal of Cancer, 2012
- PTEN restoration and PIK3CB knockdown synergistically suppress glioblastoma growth in vitro and in xenograftsJournal of Neuro-Oncology, 2010
- Elevated expression of Dickkopf-1 increases the sensitivity of human glioma cell line SHG44 to BCNUJournal of Experimental & Clinical Cancer Research, 2010
- PI3K pathway alterations in cancer: variations on a themeOncogene, 2008
- PI3K regulates pleckstrin-2 in T-cell cytoskeletal reorganizationBlood, 2006
- Purification and characterization of phosphoinositide 3-kinase from rat liver.Journal of Biological Chemistry, 1990