Nonhost resistance: Self-inflicted DNA damage by fungal DNase accumulation is a major factor in terminating fungal growth in the pea-Fusarium solani f.sp. phaseoli interaction
Author:
- Lee A. HadwigerLHLee A. Hadwiger
- 1 October 2015
- journal article
- Published by Elsevier in Physiological and Molecular Plant Pathology
- Vol. 92, 79-87
Abstract
No abstract availableKeywords
Funding Information
- Washington State Potato Commission
This publication has 31 references indexed in Scilit:
- Multiple effects of chitosan on plant systems: Solid science or hypePlant Science, 2013
- The non-host disease resistance response in peas; alterations in phosphorylation and ubiquitination of HMG A and histones H2A/H2BPlant Science, 2009
- Localization predictions for gene products involved in non-host resistance responses in a model plant/fungal pathogen interactionPlant Science, 2009
- Pea–Fusarium solani Interactions Contributions of a System Toward Understanding Disease ResistancePhytopathology®, 2008
- A Promoter from Pea Gene DRR206 Is Suitable to Regulate an Elicitor-Coding Gene and Develop Disease ResistancePhytopathology®, 2004
- Characterization of a 20 kDa DNase elicitor from Fusarium solani f. sp. phaseoli and its expression at the onset of induced resistance in Pisum sativumMolecular Plant Pathology, 2001
- Characterization of a plant-stimulated nuclease from Fusarium solaniPhysiological and Molecular Plant Pathology, 1993
- Chitosan oligomers from Fusarium solani/pea interactions, chitinase/β-glucanase digestion of sporelings and from fungal wall chitin actively inhibit fungal growth and enhance disease resistancePhysiological and Molecular Plant Pathology, 1989
- Determination of the Pore Size of Cell Walls of Living Plant CellsScience, 1979
- Chloroplasts and Mitochondria in Living Plant Cells: Cinephotomicrographic StudiesScience, 1962