Sialic acid-binding immunoglobulin-like lectin 15 (Siglec-15) mediates periarticular bone loss, but not joint destruction, in murine antigen-induced arthritis
Abstract
No abstract availableKeywords
Funding Information
- Ministry of Education, Culture, Sports, Science and Technology (23890008)
This publication has 31 references indexed in Scilit:
- Standardized nomenclature, symbols, and units for bone histomorphometry: A 2012 update of the report of the ASBMR Histomorphometry Nomenclature CommitteeJournal of Bone and Mineral Research, 2012
- The immunoreceptor tyrosine-based activation motif (ITAM) -related factors are increased in synovial tissue and vasculature of rheumatoid arthritic jointsArthritis Research & Therapy, 2012
- Siglec-15, a member of the sialic acid-binding lectin, is a novel regulator for osteoclast differentiationBiochemical and Biophysical Research Communications, 2011
- Guidelines for assessment of bone microstructure in rodents using micro–computed tomographyJournal of Bone and Mineral Research, 2010
- Myeloid DAP12-associating lectin (MDL)-1 regulates synovial inflammation and bone erosion associated with autoimmune arthritisJournal of Experimental Medicine, 2010
- Bone Microenvironment Specific Roles of ITAM Adapter Signaling during Bone Remodeling Induced by Acute Estrogen-DeficiencyPLOS ONE, 2007
- Pathological role of osteoclast costimulation in arthritis-induced bone lossProceedings of the National Academy of Sciences, 2007
- Discovery of Siglec‐14, a novel sialic acid receptor undergoing concerted evolution with Siglec‐5 in primatesThe FASEB Journal, 2006
- Interleukin‐6 modulates production of T lymphocyte–derived cytokines in antigen‐induced arthritis and drives inflammation‐induced osteoclastogenesisArthritis & Rheumatism, 2005
- Bone Loss in Rheumatoid Arthritis: Influence of disease activity, duration of the disease, functional capacity, and corticosteroid treatmentScandinavian Journal of Rheumatology, 1996