The bradycardic agent ivabradine decreases conduction velocity in the AV node and in the ventricles in-vivo
Open Access
- 1 February 2021
- journal article
- research article
- Published by Elsevier in European Journal of Pharmacology
- Vol. 893, 173818
Abstract
No abstract availableKeywords
Funding Information
- Austrian Science Fund (P210006-B11, W1232-B11)
This publication has 58 references indexed in Scilit:
- Early repolarization in mice causes overestimation of ventricular activation time by the QRS durationCardiovascular Research, 2012
- Cardiac Safety Implications of hNav1.5 Blockade and a Framework for Pre-Clinical EvaluationFrontiers in Pharmacology, 2012
- Intraventricular Conduction Delay in a Standard 12-Lead Electrocardiogram as a Predictor of Mortality in the General PopulationCirculation: Arrhythmia and Electrophysiology, 2011
- Voltage-Gated Ion Channel Dysfunction Precedes Cardiomyopathy Development in the Dystrophic HeartPLOS ONE, 2011
- Deep bradycardia and heart block caused by inducible cardiac-specific knockout of the pacemaker channel gene Hcn4Proceedings of the National Academy of Sciences, 2011
- Twenty-five years in the making: flecainide is safe and effective for the management of atrial fibrillationEP Europace, 2010
- In vivo studies of Scn5a+/− mice modeling Brugada syndrome demonstrate both conduction and repolarization abnormalitiesJournal of Electrocardiology, 2010
- Beneficial effects of delayed ivabradine treatment on cardiac anatomical and electrical remodeling in rat severe chronic heart failureAmerican Journal of Physiology-Heart and Circulatory Physiology, 2009
- ON THE MECHANISM OF CARBAMAZEPIN-INDUCED ANTINOCICEPTION IN THE FORMALIN TESTInternational Journal of Neuroscience, 2006
- Novel If Current Inhibitor Ivabradine: Safety Considerations2006