Molecular pathways of pain: Fos/Jun-mediated activation of a noncanonical AP-1 site in the prodynorphin gene
Authors:
- Josér. NaranjoJosér. Naranjo
- Britt MellströmBMBritt Mellström
- Matilde AchavalMAMatilde Achaval
- Paolo Sassone-CorsitPSPaolo Sassone-Corsit
Abstract
No abstract availableKeywords
This publication has 45 references indexed in Scilit:
- The regulation and function of c-fos and other immediate early genes in the nervous systemNeuron, 1990
- Regulation of Proenkephalin by Fos and JunScience, 1989
- The oncogene jun and nuclear signallingTrends in Biochemical Sciences, 1989
- Fos and jun: The AP-1 connectionCell, 1988
- Increased staining of immunoreactive dynorphin cell bodies in the deafferented spinal cord of the ratNeuroscience Letters, 1988
- Purified transcription factor AP-1 interacts with TPA-inducible enhancer elementsCell, 1987
- Phorbol ester-inducible genes contain a common cis element recognized by a TPA-modulated trans-acting factorCell, 1987
- Prodynorphin gene expression is enhanced in the spinal cord of chronic arthritic ratsNeuroscience Letters, 1987
- Endogenous Opioids: Biology and FunctionAnnual Review of Neuroscience, 1984
- Isolation and structural organization of the human preproenkephalin B geneNature, 1983