Neutrophil-derived ROS contribute to oxidative DNA damage induction by quartz particles
Authors:
- Anton WesselsAWAnton Wessels
- Roel P.F. SchinsRSRoel P.F. Schins
- 1 December 2010
- journal article
- research article
- Published by Elsevier in Free Radical Biology and Medicine
- Vol. 49 (11) , 1685-1693
Abstract
No abstract availableKeywords
This publication has 47 references indexed in Scilit:
- The pro-inflammatory effects of low-toxicity low-solubility particles, nanoparticles and fine particles, on epithelial cells in vitro: the role of surface areaOccupational and Environmental Medicine, 2007
- Heme Oxygenase-1, a Potential Biomarker of Chronic Silicosis, Attenuates Silica-induced Lung InjuryAmerican Journal of Respiratory and Critical Care Medicine, 2006
- Expression of Heme Oxygenase‐1 in the Lungs of Rats Exposed to Crystalline SilicaJournal of Occupational Health, 2006
- Signaling Pathways Controlling The Production Of Inflammatory Mediators in Response To Crystalline Silica Exposure: Role Of Reactive Oxygen/Nitrogen SpeciesFree Radical Biology and Medicine, 2004
- Inhaled particles and lung cancer. Part A: MechanismsInternational Journal of Cancer, 2004
- Chronic obstructive pulmonary disease due to occupational exposure to silica dust: a review of epidemiological and pathological evidenceOccupational and Environmental Medicine, 2003
- Oxidant-induced DNA damage by quartz in alveolar epithelial cellsMutation Research - Genetic Toxicology and Environmental Mutagenesis, 2002
- Oxidative stress and regulation of glutathione in lung inflammationEuropean Respiratory Journal, 2000
- Effects of particle exposure and particle-elicited inflammatory cells on mutation in rat alveolar epithelial cellsCarcinogenesis, 1997
- ESR evidence for the hydroxyl radical formation in aqueous suspension of quartz particles and its possible significance to lipid peroxidation in silicosisJournal of Toxicology and Environmental Health, 1988