Pathogenesis of indirect (secondary) acute lung injury
- 1 February 2011
- journal article
- review article
- Published by Taylor & Francis in Expert Review of Respiratory Medicine
- Vol. 5 (1) , 115-126
Abstract
At present, therapeutic interventions to treat acute lung injury (ALI) or acute respiratory distress syndrome (ARDS) remain largely limited to lung-protective strategies, as no real molecular–patho...Keywords
This publication has 134 references indexed in Scilit:
- Therapeutic accessibility of caspase-mediated cell death as a key pathomechanism in indirect acute lung injury*Critical Care Medicine, 2010
- Molecular determinants of endothelial transcytosis and their role in endothelial permeabilityAmerican Journal of Physiology-Lung Cellular and Molecular Physiology, 2007
- Fas-induced Pulmonary Apoptosis and Inflammation during Indirect Acute Lung InjuryAmerican Journal of Respiratory and Critical Care Medicine, 2007
- Pulmonary and extrapulmonary acute respiratory distress syndrome: are they different?Current Opinion in Critical Care, 2005
- Differential Response of Human Lung Epithelial Cells to Fas-Induced ApoptosisThe American Journal of Pathology, 2004
- Pulmonary and extrapulmonary acute respiratory distress syndrome are differentEuropean Respiratory Journal, 2003
- Influence of direct and indirect etiology on acute outcome and 6-month functional recovery in acute respiratory distress syndromeCritical Care Medicine, 2001
- Fas (CD95) Induces Alveolar Epithelial Cell Apoptosis in VivoThe American Journal of Pathology, 2001
- Impairment of endothelial-dependent pulmonary vasorelaxation after mesenteric ischemia/reperfusionSurgery, 1996
- Adult Respiratory Distress Syndrome in Patients with Severe NeutropeniaNew England Journal of Medicine, 1986