Adaptation to moderate hypoxia protects cortical neurons against ischemia–reperfusion injury and excitotoxicity independently of HIF-1α
- 1 August 2011
- journal article
- research article
- Published by Elsevier in Experimental Neurology
- Vol. 230 (2) , 302-310
Abstract
No abstract availableKeywords
Funding Information
- American Heart Association
This publication has 37 references indexed in Scilit:
- Selective Inhibition of Hypoxia-Inducible Factor (HIF) Prolyl-Hydroxylase 1 Mediates Neuroprotection against Normoxic Oxidative Death via HIF- and CREB-Independent PathwaysJournal of Neuroscience, 2009
- Reperfusion accelerates acute neuronal death induced by simulated ischemiaExperimental Neurology, 2007
- Hypoxia Inducible Factor Prolyl 4-Hydroxylase Enzymes: Center Stage in the Battle Against Hypoxia, Metabolic Compromise and Oxidative StressNeurochemical Research, 2007
- Key Note LectureStroke, 2007
- The Transcriptional Activator Hypoxia Inducible Factor 2 (HIF-2/EPAS-1) Regulates the Oxygen-Dependent Expression of Erythropoietin in Cortical AstrocytesJournal of Neuroscience, 2006
- Inhibition of NGF deprivation–induced death by low oxygen involves suppression of BIMEL and activation of HIF-1Journal of Cell Biology, 2005
- Translation of Ischemic Preconditioning to the PatientStroke, 2004
- Hypoxia-Induced Stroke Tolerance in the Mouse Is Mediated by ErythropoietinStroke, 2003
- Evaluation of brain tissue O2 supply based on results of PO2 measurements with needle and surface microelectrodesJournal of the Autonomic Nervous System, 1996
- Neuroprotection from ischemic brain injury by hypoxic preconditioning in the neonatal ratNeuroscience Letters, 1994