Remodeling of Interstrand Crosslink Proximal Replisomes Is Dependent on ATR, FANCM, and FANCD2
Open Access
- 1 May 2019
- journal article
- research article
- Published by Elsevier in Cell Reports
- Vol. 27 (6) , 1794-1808.e5
Abstract
No abstract availableKeywords
Funding Information
- National Institute on Aging (Z01-AG000746-08)
- National Natural Science Foundation of China (31871365)
- National Natural Science Foundation of China (21708007)
- National Natural Science Foundation of China (CA190635)
- National Natural Science Foundation of China (pDC6914)
- National Institutes of Health
This publication has 81 references indexed in Scilit:
- Involvement of SLX4 in interstrand cross-link repair is regulated by the Fanconi anemia pathwayProceedings of the National Academy of Sciences, 2011
- The FANCM/FAAP24 Complex Is Required for the DNA Interstrand Crosslink-Induced Checkpoint ResponseMolecular Cell, 2010
- A Histone-Fold Complex and FANCM Form a Conserved DNA-Remodeling Complex to Maintain Genome StabilityMolecular Cell, 2010
- FANCM: fork pause, rewind and playThe EMBO Journal, 2010
- Repair of Laser-localized DNA Interstrand Cross-links in G1 Phase Mammalian CellsJournal of Biological Chemistry, 2009
- The Fanconi Anemia Protein FANCM Is Controlled by FANCD2 and the ATR/ATM PathwaysJournal of Biological Chemistry, 2009
- Mechanism of Replication-Coupled DNA Interstrand Crosslink RepairCell, 2008
- FANCM of the Fanconi anemia core complex is required for both monoubiquitination and DNA repairHuman Molecular Genetics, 2008
- The replication kinase Cdc7‐Dbf4 promotes the interaction of the p150 subunit of chromatin assembly factor 1 with proliferating cell nuclear antigenEMBO Reports, 2006
- A human ortholog of archaeal DNA repair protein Hef is defective in Fanconi anemia complementation group MNature Genetics, 2005